Fig. 5. Electron microscopic feature of cardiomyocytes at 24-hour following MI. A, C, E, G and I: Representative ultrathin section of cardiomyocyte from GAL-3 wild MI group showing A, damaged myofibrils (thin arrow) and damaged mitochondria (curved arrow). C, dilated rough endoplasmic reticulum (arrowhead), nuclear chromatin lysis (thin arrow), damaged microorganelle in a lysosome (thick arrow), and damaged mitochondria (curved arrow). E, Pyknotic nucleus with irregular nuclear membrane as early apoptotic nuclear changes in cardiomyocytes. G, two apoptotic bodies (arrowhead). I, apoptotic bodies (arrowhead) and one apoptotic nuclear changes (thin arrow). B, D, F, H and J: Representative ultrathin section of cardiomyocyte from GAL-3 KO MI group showing B, dilated rough endoplasmic reticulum (arrowhead), damaged mitochondria (curved arrow), lysosomes (thick arrow), damaged myofibrils (thin arrow), apoptotic cell (double arrowhead). D, damaged myofibrils (arrowhead), damaged mitochondria (curved arrow)and damaged sarcolemmal membrane (thin arrow). F, damaged myofibrils (arrowhead), damaged mitochondria (curved arrow), lysosomes (thick arrow), and apoptotic bodies (arrowhead). H, multiple apoptotic bodies (arrowhead) and apoptotic cells (thin arrow). J, multiple apoptotic bodies (arrowhead) and apoptotic cells (thin arrow).